Alzheimer's disease is a chronic neurodegenerative disease that destroys brain cells, causing memory and thinking skills to deteriorate over time. Alzheimer's disease is not a normal part of ageing and is irreversible.
According to university research, a link has been made with 5 food ingredients that accelerate the development of the disease. You should avoid eating them, they are the following:
- Popcorn because it has flavour packets that contain chemicals that are absorbed by the brain and alter it. (look at MGS)
- Sweeteners like aspartame, Splenda (sucralose), saccharin, Polydextrose, etc.
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Meats processed foods, such as cold meats, peperoni, Bologna
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Red meat : Red meat increases the risk of developing Alzheimer's disease. The iron in this food promotes oxidative stress, in other words an imbalance between the excessive amount of free radicals and antioxidants.
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Milk products: Too much dairy products such as butter, cream and cheese cause an accumulation of fatty bodies in the brain which accelerates the risk of worsening Alzheimer's disease.
- Aluminium: The concentration found of Aluminium is higher in the brains of people who died from Alzheimer's than from other causes of death. Unfortunately, Aluminium is everywhere. It's in drinking water, supplements, antacids, deodorants, cans, foils and is commonly used in cookware. It is suggested to limit the use of aluminium utensils and pots or to take food supplements, such as vitamins, that contain them.
- Diacetyl: And diacetyl is bad news for our memory, It causes beta-amyloid clumping with is a signifiant indicator of Alzheimer's
- Monosodium glutamate (MSG) and other sources of glutamate in foods: Glutamate is an amino acid that is part of the composition of food proteins of animal and plant origin. Glutamate is naturally present in many foods we eat every day, including: Grapes tomatoes the mushrooms certain cheeses, such as Parmesan and Roquefort fruit juices (such as grape juice) and tomato juice. Glutamine is a related amino acid that occurs naturally in high concentrations in certain foods. It transforms into glutamate in the presence of glutaminase, an enzyme added to foods. Glutaminase can be used in wheat flour, baked goods, pasta, hydrolysed proteins, certain egg products, yeast extracts and certain flavour preparations.
Poor sleep quality
Researchers continue to study this risk factor. Indeed, sleep is important because it allows the brain to be cleansed of toxic proteins formed during the waking phase (by the “glymphatic” system). Having poor sleep quality could impair these cleansing processes. And therefore contribute to the accumulation of brain damage from Alzheimer’s disease. Dr. Geraldine Rauchs demonstrated that difficulty falling asleep is linked to the formation of amyloid plaques in the brain. Fortunately, it is possible to act on the quality of sleep! Not with medications but with other methods such as exposure to light, physical activity, meditation, cardiac coherence, etc.
Solutions:
Here are some possible solutions to counter Alzheimer's disease:
- The practice of appropriate and regular physical activity
- A healthy diet ; it is suggested to use the Mediterranean diet for UK people;
- Mental stimulation supported by leisure activities;
- Maintaining social and friendly ties
- Sleep well
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Drink a glass of Kombucha with fermented ginger each day. This drink, may be not available in the market, so you will have to do it homemade. But the rewards are very good, a research shown goods results to counter the progression of the Alzheimer's disease by more than 88.3 % and make you live a more happy life for the next 10 to 15 years without suffering too pronounced dementia.
Promise of Lithium Orotate
Alzheimer's disease, a heartbreaking condition that erodes memory and identity, may have found an unlikely opponent in the form of lithium, a metal often overlooked for its potential benefits on brain health. Research conducted over the past decade at Harvard Medical School has unveiled a significant connection between lithium deficiency in the brain and the onset of Alzheimer’s. More strikingly, scientists have shown that lithium orotate supplementation can reverse memory loss in mouse models affected by the disease. This groundbreaking revelation challenges long-held assumptions about Alzheimer’s treatment and paves the way for potential new therapies.
Traditionally, lithium is known for its use in high doses to manage bipolar disorder; however, these recent findings suggest that in tiny, biologically relevant doses, lithium acts much like an essential nutrient akin to vitamins. This discovery holds critical implications, particularly as we confront a global rise in dementia cases. It is essential to explore the intricacies of this research, its potential impacts, its limitations, and what it means for the millions of individuals and families grappling with the relentless progression of memory loss.
Extensive analyses have confirmed that lithium naturally exists in the human brain at low yet vital levels, much like iron or vitamin C. This was an unexpected revelation, as the specific role of lithium in maintaining neuronal health was previously unrecognized. Recent analysis of brain tissue from healthy individuals, patients with mild cognitive impairment, and those with Alzheimer's disease has led to a remarkable discovery. Lithium is the only metal that shows a significant decrease in concentration at the initial signs of Alzheimer's. This decline is significant and not coincidental, as lithium does not evaporate. Researchers have identified a surprising and detrimental mechanism: amyloid plaques behave like sponges, absorbing surrounding lithium. This absorption deprives nearby neurons of an essential element crucial for their proper functioning, contributing to neurodegeneration.
Studies tracking lithium levels in brain samples from patients at different stages of Alzheimer’s have demonstrated a notable decline starting from the early symptoms of the disease. This lithium deficit disrupts neuronal communication, accelerates neurodegeneration, and fosters toxic amyloid plaque deposits, integral to the pathology of Alzheimer's. Ironically, these amyloid plaques absorb lithium, perpetuating a vicious cycle that further deprives the brain of this essential mineral. This cycle may account for the swift cognitive decline observed in patients.
To address this critical deficiency, researchers have turned to lithium orotate, an innovative form that navigates the obstacles posed by amyloid plaques. When injected at low, physiological doses, lithium orotate effectively restores normal lithium levels in the brains of affected mice. The results are nothing short of remarkable: memory loss is reversed, brain lesions diminish, and cognitive abilities are significantly restored. This marks a pioneering advance in Alzheimer's research, signalling a potential shift from merely managing symptoms to genuinely restoring brain health. Notably, in older mouse models with advanced disease stages, lithium orotate supplementation has shown the capacity to halt and even reverse damage, suggesting that lithium may be a curative agent capable of rehabilitating impaired brain chemistry.
The challenge has been that lithium carbonate, the traditional treatment used in psychiatry for bipolar disorder, also gets trapped by amyloid plaques. Fortunately, researchers identified lithium orotate, a specific salt that seems to evade this absorption.
In a pivotal series of animal tests, researchers examined this hypothesis using mice genetically predisposed to Alzheimer's. They found that a lithium-deficient diet accelerated all markers of the disease: plaque formation, accumulation of abnormal tau proteins, and memory decline. However, restoring normal lithium levels provided a protective effect. In supplemented mice, brain structure was maintained and cognitive functions remained intact, as if the cells regained their resilience against the disease's progression.
When this compound was administered to mice already affected by Alzheimer's, even in advanced stages, the results were impressive. It not only slowed the accumulation of plaques and tau proteins but also reduced brain inflammation and restored memory capacity. Remarkably, these results were achieved with doses a thousand times lower than those typically used in psychiatric treatments, significantly reducing the risks of toxicity to the kidneys or thyroid.
This significant discovery, published in the journal *Nature*, has been met with excitement in the scientific community. However, researchers emphasize the need for caution. While results in mice are promising, they must be validated through rigorous clinical trials in humans before any applications can be made. Self-medicating with lithium poses considerable risks, especially at higher doses that can lead to toxicity, particularly in vulnerable elderly populations. Although initial findings suggest that lithium orotate, at very low doses, has a favourable safety profile, further investigation is essential to confirm this.
These findings extend beyond therapeutic implications, opening up exciting prospects for diagnosis and prevention. In the future, we could consider screening at-risk individuals by measuring lithium levels in the blood, even before any clinical symptoms arise. This approach also suggests a new public health strategy: could maintaining stable and sufficient lithium levels throughout life help delay or even prevent the onset of Alzheimer's disease? This intriguing possibility offers immense hope for future research and prevention efforts.
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